Moving to a new place can be nerve racking experience or a pleasant Out Of State Moving Companies in Bryanston one. It basically depends on how you choose it to be. Moving and packing go hand in hand and how you pack ultimately determines how your moving in experience to a new place finally concludes. You should focus on making things smoother rather than doing everything at the eleventh hour. Steps like getting together packing tools like padding, tapes, boxes, markers etc., cleaning the house for important items, and making reservations with the movers should be taken well before time. Similar steps which should be taken care of well in advance are discussed below –
Gather All the Goods and Items
Gather all the goods and items you need to pack in one place. It often happens that Removalists important things are left behind and that is something you would not like when you are amidst unpacking in your new house/office.
Ready the Supplies and Boxes
Before you actually start packing your stuff, you need to make sure that you have all the pre-requisites such as boxes of different sizes, markers, duct tapes, and padding tools. It is very important that you have these in place before you start packing as it can be a real inconvenience getting these things later on.
Prepare Important Paperwork for the Move
Prepare a proper paperwork wherein you would have a detailed list of things that need to pack as well as the things that you have packed. It would come in very handy once you start unpacking.
Start Packing in Advance
It is always advised to start the packing process in advance. It not only saves you precious time but also reduces the hassles in the end. The lesser the hassles, Furniture Removal Truck the lesser the errors and mistakes. Even the best movers would advise you to do the same.
Use Proper Padding
Simply packing things is not enough. You need to make sure that you make proper provisions for the protection of the things you have packed. Proper padding is also important because you never know what is going to happen to your goods and how are they going to be treated. Sudden and uncertain accidents may damage your goods for good. It is thus, always advised to provide proper protection to your packed items. This can be done by bubble wraps or old and used clothes. You can even use Thermocol to add more protection to delicate items.
Take Pictures of Electronic Set-Ups
Electronic set ups can be hard to put back the same way as they were set up before. It often happens that people are unable to set up electronics the exact same way after they have moved into a new place. It is therefore highly recommended that you take photographs of such complicated electronic set ups so that you can late refer to these pictures while setting up again in your new house/office.
Pack in Separate Boxes
While packing your stuff, it is important that you use different boxes and that too of various sizes. It is imperative to do so as this will help you a lot while you unpack. Packers and movers would advise you the same as it is very convenient to unpack different things from different boxes as compared to unpacking everything from one single box.
Pack on An “Open – First” Basis
Last but definitely not the least, always pack your stuff as per ’Open – First” basis. While you pack, always put in stuff which you will need last. This way, when you start unpacking, you will get your hands on things that you need immediately.
IntroductionOn the June 1st 1960, Bazil Thorne won 100, 000 pounds in the tenth Opera House Lottery. Little did he or his wife Freda know, this substantial win would affect his family in a way no body would ever dream of and forever change Australia's way of life. A Strange VisitorA few weeks before Graeme was murdered, Mrs Thorne answered a knock at her front door. A man of European appearance asked Mrs Thorne if she knew a man by the name of Mr Bognor. Mrs Thorne said that she didn't know of anybody by that name and closed the door. The "Stocky" ManIn the days leading up to the kidnapping and murder of Graeme Thorne, several people noticed a "stocky" built man of olive complextion sitting in the park directly across the road from the Thorne house. The description of the man was very similar to the description Mrs Thorne had given of the man who had knocked on her door a few weeks before. July 7th, 1960Early on the morning of July 7th, a man by the name of Cecil Denmeade noticed an iridescent blue Ford model car parked on an intersection less than 100m from the Thornes' house. Shortly after 8:30am, Graeme Thorne left for school. He was to walk to the nearby corner of O'Brien and Wellington Streets, where he was to be picked up by family friend Phyllis Smythe and her two sons and taken to school at Scots College in the nearby suburb of Bellevue Hill. When Mrs Smythe arrived and noticed Graeme was not waiting for her, she sent one of her sons to go and look for him. When he couldn't be found, Mrs Smythe drove straight to the Thorne's house. Upon learning that her son was not at his usual pick up spot, Mrs Thorne phone the Bondi police to report her son missing. Shortly after Mrs Thorne contacted police, Sergeant Larry O'Shae arrived at the Thorne house to begin investigating. About 9:40am, Mrs Thorne received a phone call from a male with a foreign accent, she handed the phone to Sergeant O'Shae who told the caller he was the child's father. The caller demanded 25, 000 pounds before 5pm that day for the safe return of the boy. Sergeant O'Shae was told if the money was not delivered the boy would be "fed to the sharks". Realizing the seriousness of the situation Sergeant O'Shae contacts Bondi police station and asks for reinforcements. Detective Sergeant Lloyd Noonan arrives a short time later. While the police had been trying to keep the news of the kidnapping out of the newspapers, crime reporter Bill Jenkings found out and the story appeared on the front page of the Sydney Daily Mirror, with the headline "I'll feed him to the sharks". Mr Thorne, who was away on a business trip arrived back at Sydney airport later that evening, he was met by police and informed that his son had been kidnapped. At around 8:30pm, Mr Thorne appeared on television, making a desperate plea for the safe return of his son. During the appeal he said "All I can say, the person that's got him - if he's a father and got children of his own - well, for God's sake, send him back in one piece." (CI-A Book 2 page 3) The caller phoned again around 9:45pm and after checking that the money was available, gave instructions that the money be placed in two paper bags, but hung up without any further instructions and made no further contact or demands. The New South Wales government offered a 5000 pound reward for any information leading to the arrest and conviction of Graeme Thorne's kidnapper and newspapers offered a further 15, 000 pounds. The Search For Graeme Gets UnderwayA massive police operation was set up, led by Detective Sergeant Ernie Freeman. Police contacted Cecil Denmeade as they were doubtful of the detailed description of the car he had provided. Mr Denmeade recalls "They picked me up in the evening, in the dark, and took me round the backstreets of Bondi and showed me cars, to try to get me to recognize the same model Ford as I'd reported" (CI-A book 2 page 6) Convinced of Mr Denmeade's description, Detective Sergeant Doyle sent 25 police officer to the Department of Transport to search and record all of the 1955 Ford Customlines. After almost a week, the police had recorded over four thousand cars matching the make and model. Each of these were located and if possible, the owner was interviewed. Another large group of police officers were given the task of door-knocking the suburb of Bondi. All major roads, as well as, airports and seaports were put under surveillance. On July 8th, Graeme's school case was discovered in bushland on the other side of Sydney Harbour, about 20km north of Bondi. Police moved to the scene and set up a makeshift headquarters at the location. The following day, July 9th, hundreds of police begin to search the area were the school case was located. They are joined by Navy helicopters, Army commandos and police skindivers. Specially trained police sniffer dogs are brought in from South Australia to assist with the search. The search lasted several day, but failed to turn up anything. Following LeadsThere were several sighting of Graeme reported to police from all over Australia. The police pain-stakingly recorded and investigated each one, no matter how far-fetched the lead seemed. Each lead was discounted. Of course, there were people that were contacting the police clamming they either had Graeme, or had "psychic" visions and knew were Graeme was in order to receive not only the reward, but also the ransom. The Graeme Thorne case resulted in the largest police file ever produced in Australia. August 16th 1960On August 16th, 1960 everybody's worst fears were realised. Several children playing in a vacant lot located in the northern Sydney suburb of Seaforth discovered a small bundle wrapped in a blanket underneath a rock ledge. They informed one of the children's mothers and were told not to touch it and to wait until their fathers arrived home from work, so the men could go over and investigate further. When the fathers arrived home, the group of men walked across the lot to the rock ledge and discovered the body of Graeme Thorne. Bazil and Freda Thorne learned of their son's death while watching television, moments before Detective Sergeant Ken Baret and the church minister arrived to inform them. Detective Sergeant John Snowden of the police scientific bureau was called to help investigate the scene. The following morning, August 17th, an autopsy was conducted. The conclusion was Graeme was either killed by blows inflicted to the head or strangulation. Forensic InvestigationThe rug Graeme's body was covered with was identified as an Onkaparinga brand rug. It was taken to the police scientific division. Detective Sergeant Snowden and other member of the unit used tweezers to collect hairs, fibres, seeds and other debris from the rug. Government microbiologist, Dr Cameron Cramp received and analized the hair and fibre samples collected from the rug. He concluded that hairs on the rug were a 100% match to those belonging to a Pekingese dog. Meanwhile, soil samples taken from Graeme's clothing were sent to Horace Whitworth, the curator of the Geological and Mining Museum in Sydney. Soil and vegetable matter were also sent to Professor of Plant Pathology Neville White. Other foliage removed from the rug were sent to botanist Dr Joyce Vickey at Sydney's Royal Botanical Gardens. Dr Vickey's examination of the foliage was able to give the police their second break in the case. Crucial Evidence RevealedThe forensic analysis reviled the following The two shrubs were identified as 1. Dwarf Blue Sawara Cypress 2. Blue Ice Cypress Combined with the information police already had 1. A male with a foreign accent 2. A iridescent blue 1955 Ford Customline sedan Keeping this information in mind, police painstakingly conducted a house to house door-knock of all of the houses in and around the area where the body had been discovered. While the two shrubs were common by themselves in the area, the combination of both was not. That is until the police discovered one house in the suburb of Clontaft, located about 3km away from where the body had been found. Police immediately became interested in the residence living in this house. The Investigation Into Stephen Bradley BeginsPolice conducted a search of the property in Clontaft, which revealed the soil under the house contained fragments of pink mortar and directly in front of the garage were the two plants identified during the analysis. Police also discovered that the house, which was recently sold, was previously owned by a Hungarian by the name of Stephen Leslie Bradley and his wife Magda. The also discovered that Stephen Bradley had arranged for furniture removalists to be at the house at 11am the day Graeme disappeared. They also discovered that Magda and two of their three children left for the city shipping terminal in a taxi at 10am on that day. They also found that Stephen Bradley was the owner of a blue 1955 Ford Customline, which was recently sold to a caryard in the suburb of Granville. Analysis of the CarWith the car located, police began to examine it. In the boot they found a hairbrush which contained a number of dog hairs, which matched the hairs found on the rug and parts of Graeme's clothing. There was also human hairs found on the floor of the car. Police also received information that Stephen Bradley had owned a second car, a Googomobile. This car was also found and examined. This examination also revealed human and dog hairs, which matched those found during the analysis. Further DiscoveriesBoth human and animal hair was also discovered in an apartment in the suburb of Manley, where Stephen Bradley had lived shortly before leaving Australia in late September 1960. Hair was also discovered in a vacuum cleaner and carpet sweeper owned by Stephen Bradley and sold in September 1960. Police also recovered a crumpled length of thirty-five mm black and white film which was sent for analysis, they discovered photos of the Bradley family sitting on a rug that was identical in pattern to the one Graeme's body was wrapped in. The Bradley's dog was found at a Sydney veterinary hospital, samples of hair were taken and it was confirmed that these hairs were a match with the hairs found on the rug. Police found two pieces of twine in the yard of the house in Clontaft, as well as from the apartment in Manley and two items of furniture, which had also been sold. This twine was examined and matched to the twine that had been tied around Graeme's ankles. Further Examination of the RugA pale-blue rug tassel, found under the house in Clontaft was confirmed as matching the tassels on the rug Graeme had been wrapped in. To further confirm the Bradley's owned a rug like the one found came from a person in Melbourne, he stated he had given a rug similar in type and colour to Mrs Bardley sometime during 1955. Finding Stephen BradleyWith the confirmation that Stephen Bradley was indeed the person the police were looking for, police set about locating him. The learned that he and his family were onboard a ship named "Himalaya" which was headed for London, England. Detectives alerted the ships captain and Stephen Bradley was put under surveillance while police submitted extradition papers. Stephen Bradley was arrested shortly after the ship docked in Colombo, Sri Lanka. At first he proclaimed his innocence, but during return flight to Sydney he told the police which were escorting him back to Australia he had indeed committed the crime. Stephen Bradley's written confessionI red in the newspaper that Mr. Thorne won the first prise in The Operahouse Lottery. So I desided that I would kidnap his son. I knew ther address from the newspaper, and I have got their phone number from the telephone exchange. I went to the house to see them. I have asked for someone but cannot remember what name. Mrs. Thorne said she did not know that name and she told me to enquire in the flat upstairs. I went upstairs and I seen the woman there. I have done this because I though that the Thornes will check up. I went out and watched the Thorne boy leaving the house and seen him for about three mornings and I have seen where he went. And one morning I have followed him to the school at Bellevue Hill. One or two mornings I have seen a womman pick him up, and take him to the school. On the day we moved from Clontarf I went out to Edward Street. I parked the car in a street I don’t know the name of the street it is off Wellington Street. I have got out from the car, and I waited on the cornor, until the boy walked down to the car. I have told the boy that I am to take him to the shool. He sed why, where is the lady. I sed she is sick and can not come today. Then the boy got in the car, and I drove him around for a while, and over the harbour bridge. I went to a public phone box near the spit bridge and I rang the Thornes. I talked to Mrs. Thorne and then to a man who sed he was the boys father. I have asked for £25,000 from the boys mother and father. I told them that if I don’t get the moneys I feed him to the sharks, and I have told them I ring later. I took the boy in the car home to Clontarf and I put the car in my garage. I told the boy to get out of the car to come and see another boy. When he got out of the car I have put a scarf over his mowth, and put him in the boot of the car, and slamed the boot. I went into my house and the Furniture Removalist came, a few minutes after. When it was nearly dark, I went to the car and found the boy was dead. That night I tied the boy up with string and put him in my rug. I put the boy in the boot of the ford car again, and them I throw his case and toys out near Bantry Bay, and I put the boy on a vacant lotmount near the house I went to see with an Estate Agent, to buy it some time before. Signed : S. L. Bradley Witness: J. H. Bateman - Detective Sergeant 2nd Class, C.I. Branch, 19-11-1960, 10am (Extract from the Australian Police Journal written by Detective Sergeant A F Clarke (NSW Police), July 1963, reprinted September 1996) Stephen Bradley Stands TrialBetween March 20th and 29th 1961, Stephen Bradley stood trial for murder at the Central Criminal Court in Sydney. During the trial Stephen Bradley pleaded not guilty and stated that the confession he had given had been dictated to him by the police, he also stated that he wrote the confession as he feared for the safety of his wife and children. Stephen Leslie Bradley was found guilty of murder and sentenced to life imprisonment, which was the maximum penalty for murder in New South Wales. Stephen Bradley died of a heart attack in 1968. Changing The LawAs a direct result of the Graeme Thorne case, Australian Law was changed to allow lottery winners to protect themselves, allowing them to decline having their names and addresses published. This case also led the way in the pioneering of forensic investigation.
Several Autoimmune DisordersDid you know there are more than 80 yypes of autoimmune disorders? No, I promise not to list all of them. I will briefly cover the more common autoimmune diseases that I haven’t written about in the past. I have recently written about Rheumatoid Arthritis, Sjogren’s Disease and several hubs on Lupus so you can read detailed information on those diseases. In the normal person, the body’s immune response protects them from invading diseases and infections. In a person with an autoimmune disorder things have gone haywire. Your body doesn’t recognize invaders from your healthy tissue, so your white blood cells or T cells attack healthy organs. They can virtually affect every part of your body. These diseases usually attack women, particularly African-American and Native-American women. Many autoimmune disorders have similar symptoms, which makes diagnosis that much more difficult. Often this process is frustrating as your first symptoms are fatigue, muscle aches and low fever. The diseases sometimes flare-up and sometimes go into remission. Most autoimmune diseases affect women more often than men, with higher percentages for Afro-Americans and Native-Americans people. Celiac DiseaseCeliac disease is another autoimmune disease characterized by an inappropriate immune response to dietary proteins found in wheat, rye, barley (gluten and gliadin). This response leads to inflammation of the small intestine and to damage and destruction of the villi that line the intestinal wall. These villi are projections (small folds) that increase the surface area of the intestine and allow nutrients, vitamins, minerals, fluids, and electrolytes to be absorbed into the body. When the villi are destroyed, the body is much less capable of absorbing food and begins to develop symptoms associated with malnutrition and malabsorption. When the body is exposed to the gluten and gliadin proteins, it forms antibodies that recognize and act against not only the grain proteins, but also against constituents of the intestinal villi. As long as the patient continues to be exposed to the proteins, he will continue to produce these auto-antibodies. Celiac disease is most prevalent in those of European descent, probably inherited, can affect anyone at any age. It is thought to be an inherited tendency that is triggered by an environmental, emotional, or physical event.. The possible symptoms include: abdominal pain and distension anemia bleeding tendency bloody stool bone and joint pain changes in dental enamel diarrhea fatigue greasy foul-smelling stools oral ulceration, weakness weight loss. Children with this disease may experience delayed growth and development. It is estimated that 1 in 133 people in the U. S. have celiac disease but only 3% have been diagnosed. Patients with celiac disease must follow a lifelong gluten-free diet.. Once all forms of wheat, rye, and barley have been removed from the diet the patient improves. It is important to detect and treat celiac disease as soon as possible, especially in young children. Celiac disease should be considered in infants who are not thriving, since foods with gluten are common and celiac auto-antibodies may begin to develop shortly after a child switches from milk to solid foods. In most cases the patient can lead a normal life by adhering to the diet. SclerodermaThis is in a group of rare, progressive diseases that involve hardening and tightening of the skin and connective tissues — the fibers that provide the framework and support for your body. Localized scleroderma affects only the skin. Systemic scleroderma also harms internal organs, such as the heart, lungs, kidneys and digestive tract. Scientists estimate that about 250 people per million have some form of scleroderma. The most prevalent signs of this disease include Raynaud’s phenomenon, Gastroesophageal reflux disease (GERD) which in addition to acid reflux you may have trouble absorbing nutrients, and skin changes which may include swollen fingers and hands, thickened patches of skin, particularly on the fingers; and tight skin around the hands, face or mouth. The skin will appear shiny because it is so tight. I have a friend with this disease and she must wear a restrictive binding on her left arm from the top to her wrist, which means she always wears long sleeves. That’s not much fun living in Florida. This disease affects not only your skin but also your blood vessels and internal organs; there are sub-categories defined by what area of the body is being attacked. One is called CREST and results from an overproduction and accumulation of collagen in body tissues. Native Americans get this disease 20 times more often than the general population, and it is more common with Afro-Americans as well. It occurs 4 times more often in women than men. While there is no known cause exposure to silica dust, common in coal mines and rock quarries, some industrial solvents such as paint thinners and certain chemotherapy drugs may exacerbate the disease. There is no cure and treatment is typically the same as that for lupus patients, corticosteroids, Plaquenil, and anti-inflammatory medications. PolymyositisThis is another uncommon connective tissue disease which is characterized by muscle inflammation and progressive weakness, particularly in skeletal muscles which control movement, involving those closest to the trunk of the body first. It most commonly occurs in 30-50 year old people. Signs usually develop gradually over weeks or months. The weakness is symmetrical; affecting both the left and right sides of your body, and tends to gradually worsen. Polymyositis is in a group of diseases or disorders of the muscles called inflammatory Periods of remission are possible. Treatment to strengthen muscles should start early as there is no cure. Other symptoms may include difficulty swallowing, speaking, mild joint or muscle tenderness, fatigue and shortness of breath. Treatment is corticosteroids, (which often start at very high doses then taper down), Cytoxan, cyclosporine and sometimes intravenous therapy of Immunoglobulin containing healthy antibodies from blood donors. There are several investigation drugs also being tried at the present time. These patients will need physical and speech therapy, plus a dietetic assessment. Raynaud’s DiseaseRaynaud's disease is a condition that causes some areas of your body — such as your fingers, toes, tip of your nose and your ears — to feel numb and cool in response to cold temperatures or stress. In Raynaud's disease, smaller arteries that supply blood to your skin narrow, limiting blood circulation to affected areas. It is more common in cold climates and also affects women more than men. This disease frequently is present along with other autoimmune diseases. During an attack of Raynaud's, affected areas of your skin usually turn white at first. Then, the affected areas often turn blue, feel cold and numb, and your sensory perception is dulled. As circulation improves, the affected areas may turn red, throb, tingle or swell. There are two type of Raynaud’s disease; primary and secondary. The secondary is considered the more serious disease and usually appear after 40 years of age. The causes of this disease are numerous; other autoimmune diseases, carpal tunnel syndrome, repetitive trauma, smoking, chemical exposure and certain medications including some blood pressure treatments Juvenile Rheumatoid ArthritisIts It is the most common type of arthritis of those under 16 years of age, affecting 50,000 in the U. S. It’s a chronic condition causing joint swelling, inflammation, pain, swelling, redness and stiffness. I t may also affect the eyes and internal organs. About 1/5th of patients have an enlarged spleen. There are 3 categories: Pauciarticular- about 50% of the cases are this type and most common in girls under 8 years old Polyartifular – about 30% of the cases and it affects 5 or more joints, especially in the hands and feet. Systemic – about 20% have this type and if affect both joints and internal organs. These children may have frequent fevers and rashes that come and go rapidly. Doctors diagnosis this disorder once a patient has had the symptoms for at least 6 weeks. These symptoms may include morning stiffness, limping, reluctance to move an affected joint, joint pain and swelling. Patients with systemic JRA may have intermittent fever, rash, swollen lymph nodes, and in some cases liver, spleen, (very rarely) lung involvement and eye inflammation. The cause of this autoimmune disease is also unknown. It tendency may be inherited but is believed to that a triggering event is required for it to emerge. Several types of blood tests and x-rays are done to aid diagnosis. There is no cure so treatment is to relieve the discomfort of the symptoms. It is a heart breaking disease to see children suffer. Autoimmune PancreatitisThe term "autoimmune pancreatitis" was first used in Japan in 1995 to describe a newly recognized form of chronic pancreatitis. Since then, Mayo Clinic has played a major role in identifying, describing and treating the disorder in the United States. Mayo Clinic researchers have identified 2 types of this disease including one that can develop as young as 12 years of age. It is diagnosed from several test with the most accurate being a core biopsy. Symptoms of autoimmune pancreatitis often improve dramatically after a short course of corticosteroids. But relapses are common, and some patients require additional or long-term therapy. Multiple Sclerosis This is a chronic autoimmune disease that affects the central nervous system. It causes Inflammation and the destruction of myelin. Myelin surrounds nerve fibers and acts like insulation on a wire preventing “short-circuits” that divert a nerve signal from having its desired effects. The “demyelination” process interferes with nerve impulse transmission, affects muscular control, and causes a variety of sensory, motor, and psychological symptoms. Again the cause is unknown but it is thought to be an autoimmune process triggered by a virus, environment factors and it has a genetic predisposition The first symptoms usually occur between ages 20-40. It affects women more frequently than men, is more common in Northern European Caucasians. It important to manage the symptoms of multiple sclerosis, as well as, treating the disease before permanent damage causes symptoms. Interferon, corticosteroids and several other medications are used. Wegener’s GranulomatosisThis is another uncommon autoimmune disease that affects about 1 in 20,000 people. Again there is no known cause or cure. Symptoms are due to inflammation that can affect many tissues in the body, including blood vessels (vasculitis), which makes it a very serious disease. It affects the upper (sinuses and nose), and lower (lungs), respiratory system and frequently involves the kidneys, lungs, eyes, ears, throat, skin and other body organs. Other symptoms include nasal membrane ulcerations and crusting, saddle-nose deformity, inflammation of the ear with hearing problems, inflammation of the eye with sight problems, cough (with or without the presence of blood), pleuritis, (inflammation of the lining of the lung), rash and/or skin sores, fever, lack of energy, weakness, fatigue, loss of appetite, weight loss, arthritic joint pain, night sweats, and blood in urine which may or may not be indicated by a change in urine color. Diagnosis is established by clinical and laboratory findings such as the ANCA blood test, other blood and urine tests, x-rays, and tissue biopsy. Treatment varies based on patient symptoms and disease activity. Corticosteroids and Cytoxan are the initial treatments, then Methotrexate or Azathioprine. Graves DiseaseGraves’ disease is the most common cause of overactive thyroid gland. It is caused by an auto-antibody that acts like thyroid-stimulating immunoglobulin which cause the thyroid to produce excessive hormones. It is usually seen in women over 20 and may cause symptoms and signs such as weight loss, increased appetite, hand tremors, heat sensitivity, sweating, nervousness, and in some patients, protruding eyes. Patients often have a higher heart rate and an enlarged thyroid (goiter).Radioactive iodine uptake in measured amounts is swallowed and considered a critical component for treatment. I had a friend with this disease and they finally "killed" her thyroid as she didn't respond to treatment. The symptoms made her miserable in the meantime. There Are So Many More DiseasesMy intent was to give you an overview of some of the better known autoimmune diseases. If you have one of the disease I'm sure you know a good deal more about it than what I wrote, but if you have symptoms maybe one of these descriptions will help you. Of course, you want to be seen by a doctor and remember your symptoms are real, not in your head as many of us were told before we got a diagnosis. Living with a chronic illness isn't easy but it is certainly better when you get it diagnosed and find out what you can do to improve your health. There is so much literature available on the internet, in books and I linked several hubs from other authors from Hub-pages that may be of help. I wish you all to be in the best of health!
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